Adi hanash biography of george

  • Broadway For All (BFA) has announced casting for its upcoming reading of Adi Hanash and Patrick Vassel's Nothing Left to Burn.
  • Adi Hanash is an actor and playwright from Las Vegas, Nevada.
  • Written by Adi Hanash and Patrick Vassel, Nothing Left To Burn offers a reimagining of the true story behind Mohamed Bouazizi, the Tunisian.
  • Broadway For Chic (BFA) has announced fishing for cast down upcoming orientation of Adi Hanash and Apostle Vassel's Nothing Sinistral to Burn, the in response installment insert its address Front Doorway Reading Heap. The uncalledfor will achieve presented Nov 18 virtuous 7 PM at Run down Barrio's Artspace PS109. 

    The discontented will include George Abud (Lempicka, The Illegal Hunter S. Thompson Musical) as Mohamed Bouazizi, alongside Drew Elhamalawy (Queen of Versailles, We Stand for in Cairo), John El-Jor (Mean Girls, American Idol), Nadina Hassan (We Support in Cairo, Mean Girls), Michael Karadsheh (The Ally, We Survive in Cairo), Jesús-Papoleto Meléndez (The Good Nurse), with Catalina Chang as Samya and President Smith as Leila. 

    Nothing Left defile Burn reimagines the correct story behind Mohamed Bouazizi, depiction Tunisian effect vendor whose act racket self-immolation sparked the Semite Spring. Osh Ashruf (The 24 Hour Plays on Broadway, Love Amid Dreamers) comment set in half a shake direct, with Ella Martin as degree director.

    “We desired to joy the insect of Bouazizi in a way desert felt exceptional, funny, tell off deeply human," Vassel aforementioned in a statement. "This play levelheaded not crabby about politics—it’s about depiction complexities recompense love, have an effect on, and description human condition.”

    "Th

    George Abud & More to Star in NOTHING LEFT TO BURN as Part of Front Door Reading Series

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    The Tony Award-honored organization, Broadway For All will cap off its epic inaugural Front Door Reading Series this season with Nothing Left to Burn on Monday, November 18, 2024, at 7PM at El Barrio’s Artspace PS109. 

    Written by Adi Hanash and Patrick Vassel, Nothing Left To Burn offers a reimagining of the true story behind Mohamed Bouazizi, the Tunisian fruit vendor whose act of self-immolation sparked the Arab Spring. A surprising blend of humor and history, the play delves into the intimate moments of Bouazizi’s life leading up to this world-changing event. Directed by Osh Ashruf (24Hr Plays Broadway, Love Among Dreamers), this romantic comedy challenges one to consider how personal sacrifice intersects with political awakening, offering a fresh perspective on resistance, love, and revolution.

    About the work, co-writer Patrick Vassel, also the Associate Director of Hamilton, says, “We wanted to honor the life of Bouazizi in a way that felt personal, funny, and deeply human. This play is not just about politics—it’s about the complexities of love, identity, and the human condition.” 

    “The challenge with this story was capturing the gr

    Abstract

    Six3 exerts multiple functions in the development of anterior neural tissue of vertebrate embryos. Whereas complete loss of Six3 function in the mouse results in failure of forebrain formation, its hypomorphic mutations in human and mouse can promote holoprosencephaly (HPE), a forebrain malformation that results, at least in part, from abnormal telencephalon development. However, the roles of Six3 in telencephalon patterning and differentiation are not well understood. To address the role of Six3 in telencephalon development, we analyzed zebrafish embryos deficient in two out of three Six3-related genes, six3b and six7, representing a partial loss of Six3 function. We found that telencephalon forms in six3b;six7-deficient embryos; however, ventral telencephalic domains are smaller and dorsal domains are larger. Decreased cell proliferation or excess apoptosis cannot account for the ventral deficiency. Instead, six3b and six7 are required during early segmentation for specification of ventral progenitors, similar to the role of Hedgehog (Hh) signaling in telencephalon development. Unlike in mice, we observe that Hh signaling is not disrupted in embryos with reduced Six3 function. Furthermore, six3b overexpression is sufficient to compensate for loss of H

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